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Furthermore, some genes appear to be specially selected at relapse. As an example, little clones harboring TP53 mutations generally increase and dominate the disorder soon after CIT, which clarifies the inadequate prognosis associated with these subclonal mutations.twelve,62 In addition to TP53, mutations in IKZF3 and SAMHD1 have also been recurrently https://linkalternatifmbl7714568.blogpostie.com/54034211/the-fact-about-mbl77-that-no-one-is-suggesting

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